A multicenter case-control study in Iran reported that self-reported prolonged mobile phone use was associated with higher odds of confirmed and suspected breast cancer status. The authors emphasize that the findings do not imply causation and note limitations including self-reported exposure and potential residual confounding. They call for larger prospective studies with objective exposure assessment.

This PubMed-listed paper argues that the U.S. regulatory framework for radiofrequency radiation (RFR) from wireless technologies is outdated, lacks adequate oversight and enforcement, and has not been meaningfully updated since 1996. It contends that FCC exposure limits focus on short-term, high-intensity effects and do not address long-term, low-intensity exposures, with insufficient safeguards for children, pregnancy, and other vulnerable groups. The authors also discuss alleged regulatory capture, gaps in monitoring and compliance, and propose reforms including independent research, updated safety limits, and stronger pre- and post-market surveillance.

This rat study examined acute (1-week) and chronic (10-week) exposure to 2100 MHz radiofrequency radiation (3G UMTS-like) and assessed auditory mismatch negativity (MMN) alongside biochemical and histological brain measures. The abstract reports that acute exposure was associated with reduced MMN-related electrophysiological parameters and changes in GluR2 and GFAP with observed brain ultrastructural alterations. Chronic exposure showed opposite protein trends and enhanced MMN parameters versus chronic controls, and lipid peroxidation was not significantly different.

This RF Safe article argues that the main barrier to addressing radiofrequency radiation (RFR) and other non-native EMFs is structural policy and governance failure rather than a lack of scientific evidence. It cites the 2021 D.C. Circuit decision in Environmental Health Trust et al. v. FCC as criticism of the FCC’s rationale for keeping 1996 RF exposure limits, and it points to the Radiation Control for Health and Safety Act of 1968 as a mandate for HHS to run a research-backed radiation control program. The piece also references the U.S. National Toxicology Program’s animal findings and frames the lack of further NTP RF studies as a policy shortcoming, while promoting an “S4 MITO spin” mechanistic framework and a proposed “Clean Ether Act.”

RF Safe presents “S4 MITO spin” as a proposed mechanistic framework arguing that peer-reviewed evidence can be unified to explain reported biological effects from radiofrequency radiation (RFR) and other non-native EMFs. The post highlights animal studies (notably NTP and Ramazzini) as showing carcinogenic “signals” and emphasizes non-linear dose–response patterns, asserting relevance to regulatory exposure limits. It frames the model as empirically grounded and testable, while acknowledging it is not a complete theory of all EMF effects.

RF Safe published a white paper by John Coates arguing that current wireless (RF) exposure limits focus on thermal heating while ignoring “non-thermal” biological effects reported in many studies. The piece cites animal studies (U.S. National Toxicology Program and Ramazzini Institute) and links RF exposure to outcomes such as rare tumors and declining sperm counts, and it alleges regulatory capture. It promotes Li‑Fi and other “biologically compatible” connectivity as a proposed path forward.

RF Safe argues that a single biological mechanism explains a wide range of alleged harms from real-world radiofrequency radiation, emphasizing pulsed/modulated signals. The post claims these pulses affect voltage-gated ion channels (via the S4 voltage sensor), disrupting calcium signaling and leading to health effects. It also alleges industry “cover-up” and criticizes RF exposure limits as unchanged since 1996, while referencing animal findings and a personal anecdote.

RF Safe argues that radiofrequency radiation (especially pulsed or modulated signals with low-frequency components) can alter local membrane potentials at nanometer scales where voltage-gated ion channel S4 sensors operate. It claims these shifts could change ion channel gating in immune cells, altering calcium and proton signaling, increasing oxidative stress, and promoting innate immune activation that may contribute to autoimmune-like inflammation. The piece presents a mechanistic causal chain and highlights heart and nerve tissue as potentially more susceptible due to high ion-channel density and mitochondrial content, but does not present new study data in the provided text.

This policy-focused paper contends that U.S. oversight of radiofrequency radiation from wireless technologies is outdated and insufficient, with exposure limits and testing approaches not aligned with modern long-term, chronic exposure scenarios. It emphasizes gaps in protections for children, pregnancy, vulnerable populations, workers, and wildlife, and describes limited monitoring, research, and enforcement capacity. The author proposes reforms to improve independent research, science-based limits, surveillance, and regulatory transparency.

This animal study examined whether paternal exposure to 4.9 GHz (5G) radiofrequency radiation affects male offspring in C57BL/6 mice. It reports increased anxiety-like behavior and reduced sperm quality in adult F1 males from exposed fathers, alongside reported LRGUK hypermethylation and reduced LRGUK expression in testes. The abstract reports no significant effects on depression-like behavior, learning/memory, or fertility across F1–F2 generations.

This multicenter case-control study in Iran (n=226) examined associations between mobile phone use and breast cancer outcomes in women. Reporting more than 60 minutes/day of phone conversations was associated with higher odds of confirmed invasive breast cancer and of being classified as a suspected case versus <10 minutes/day. The authors emphasize that the results do not establish causation and may be influenced by self-reported exposure and residual confounding, warranting cautious interpretation.

This review summarizes studies reporting radiofrequency radiation (RFR)-associated changes in gene expression across biological systems. Reported affected genes relate to cellular stress responses, oxidative processes, apoptosis, DNA damage detection/repair, protein repair, and neural function regulation. The authors highlight reported gene expression effects at or below 0.4 W/kg SAR and argue this challenges current guideline assumptions, while noting that not all studies find significant effects.

This animal study examined whether prenatal exposure to 3.5 GHz radiofrequency radiation (2 hours/day) affects male reproductive outcomes later in life. Male rat offspring assessed at 12 months showed multiple adverse testicular and cellular findings in exposed groups versus sham controls, including impaired spermatogenesis markers, increased abnormal sperm morphology, increased DNA damage, and increased apoptosis, with full-gestation exposure generally most pronounced. The authors interpret the results as evidence of persistent reproductive toxicity from prenatal exposure and call for further mechanistic work and precautionary actions.

This paper evaluates and critiques 12 WHO-commissioned systematic reviews and meta-analyses on RF-EMF health effects across outcomes including cancer and reproductive endpoints. It argues that serious methodological flaws and limitations in the WHO reviews prevent them from providing assurance of safety for cell phones and other wireless devices. The authors highlight reported evidence in the animal cancer review (high certainty for heart schwannomas; moderate certainty for brain gliomas) and describe dose-related adverse effects on male fertility and reproductive outcomes, including at exposure levels below current ICNIRP thresholds.

This animal study examined 2100 MHz radiofrequency radiation exposure effects on auditory brainstem responses and brain oxidative/ultrastructural markers in adult rats. The 1-week exposure group showed prolonged ABR latencies and biochemical/structural changes consistent with oxidative stress and cellular injury. The authors report no harmful effects in the 10-week exposure condition with rest days under the studied protocol.

This paper presents a mixed-method study protocol examining electromagnetic hypersensitivity (EHS) in relation to mobile phone radiofrequency radiation exposure among undergraduate students. The quantitative component aims to identify predictors of EHS using a biopsychosocial model, while the qualitative component explores individual experiences through in-depth interviews. The abstract provides study design details and sample size but does not report study results.

This exposure assessment measured outdoor RF radiation from 4G/5G base stations near 10 schools in Örebro, Sweden (October 2024). The authors report maximum levels of 10,716–68,452 μW/m² and state these are far above EUROPAEM EMF guideline ranges; two locations also showed higher peak readings with a second meter. The paper frames these findings as indicating a significant health risk, particularly for children and sensitive groups, though no health outcomes were measured in this study.

This animal study examined short- and long-term 700 MHz (lower-band 5G) radiofrequency exposure in female Wistar rats, comparing control, sham, and exposed groups. It reports no DNA damage and no change in estrous cycle length, but increased ovarian oxidative stress markers in exposed animals. Long-term exposure was associated with ovarian histopathological alterations, while estradiol and progesterone stayed within normal ranges and testosterone increased slightly but significantly.

This narrative review summarizes human and animal research on radiofrequency (RF) radiation exposure (e.g., mobile phones, Wi‑Fi, occupational sources) and male reproductive outcomes. It reports that the literature links RF exposure with reduced sperm quality and increased DNA damage, often alongside oxidative stress and other proposed biological changes. Although inconsistencies are acknowledged, the authors conclude the overall evidence suggests harmful associations and call for standardized, long-term studies and reconsideration of guidelines.

This animal study examined whether continuous prenatal exposure to 2450 MHz radiofrequency radiation (simulating Wi‑Fi) affects thyroid tissue in rat offspring at 6 months. The exposed group showed significantly increased mononuclear cell infiltration and vascular congestion in thyroid histology. TUNEL-positive cell percentage and H2A.X antibody levels did not differ significantly between groups.