A multicenter case-control study in Iran reported that self-reported prolonged mobile phone use was associated with higher odds of confirmed and suspected breast cancer status. The authors emphasize that the findings do not imply causation and note limitations including self-reported exposure and potential residual confounding. They call for larger prospective studies with objective exposure assessment.

This RF Safe commentary argues that Reuters-reported actions by HHS and FDA—launching an HHS study and removing older FDA webpages stating cellphones are “not dangerous”—should be understood as a risk-communication/scientific-integrity adjustment rather than a declaration of confirmed harm. It contends that categorical safety messaging is not justified given mixed evidence, citing the D.C. Circuit’s 2021 decision criticizing FCC reliance on conclusory FDA statements, along with selected human, animal, and mechanistic literature. The post calls for more uncertainty-aware, evidence-graded public messaging about RF exposure from phones.

This RF Safe article reports that in mid-January 2026 HHS, led by Secretary Robert F. Kennedy Jr., removed or redirected certain FDA webpages that previously conveyed strong “no-risk” conclusions about cellphone radiation. It argues the updated FDA framing emphasizes statutory duties (monitoring, testing, hazard control) and signals a shift from definitive safety messaging toward renewed inquiry, while noting that details of any planned research have not been publicly disclosed. The piece also highlights Kennedy’s past public statements alleging harms from Wi‑Fi/5G and links the policy context to the 2021 D.C. Circuit remand of FCC RF policy.

This RF Safe commentary argues that dismissing the National Toxicology Program (NTP) cellphone-radiation animal findings as “too high dose” is misleading because the NTP used multiple exposure tiers, including a lowest tier described as near regulatory relevance. It also claims FDA has removed webpages containing prior “safety conclusion” language while HHS has announced a new study on electromagnetic radiation and health effects, framing these as a meaningful shift in federal public-facing posture. The piece further points to the Ramazzini Institute animal study as suggesting similar tumor signals at lower exposure levels, while acknowledging animal studies alone do not establish human causation.

RF Safe reports that HHS confirmed plans to launch a new study on cellphone radiation and that an HHS spokesperson said the FDA removed webpages with “old conclusions” while new research is undertaken to identify knowledge gaps, including for emerging technologies. The article frames the FDA webpage changes as a meaningful shift away from categorical reassurance, while noting Reuters’ reporting that some FDA and CDC pages still state there is no credible evidence of health problems from cellphone radiation. It also links the development to the 2021 D.C. Circuit decision in Environmental Health Trust v. FCC, arguing the ruling exposed weaknesses in the FCC’s reliance on other agencies’ statements.

RF Safe reports that the U.S. FDA substantially revised its cellphone radiation webpages around January 15, 2026, removing or reducing prior language that broadly reassured the public about safety. The article argues the new framing more closely reflects the FDA’s statutory responsibilities under the Radiation Control for Health and Safety Act of 1968 (Public Law 90-602), emphasizing research, monitoring, and public information rather than definitive safety conclusions. It also links the change to a reported HHS announcement of a new study and portrays the update as a shift toward greater transparency, while noting some safety language may remain on the page.

RF Safe recounts a timeline of FTC actions and consumer guidance targeting phone “radiation shield” stickers/patches that claimed large reductions in exposure, arguing these products can create a false sense of security. The post cites the FTC’s February 2002 enforcement actions and consumer alert, including references to Good Housekeeping Institute testing that reportedly found the products did not reduce exposure. RF Safe also claims it warned about such scams earlier (late 1990s), framing this as its own account rather than an FTC-attributed origin story.

RF Safe argues that “no effect” findings in some RF exposure studies should be interpreted as meaningful negative controls rather than as evidence that RF has no biological effects. The post presents RF Safe’s “S4–Mito–Spin” framework, claiming certain skin cell types (fibroblasts and keratinocytes) are predicted to be relatively resistant to non-thermal RF effects, so null results in these cells can be consistent with the model. It cites in-vitro studies at 3.5 GHz (5G-modulated) reporting no changes in ROS measures, stress responses, or UV-B DNA repair kinetics under specified SAR conditions, and frames these nulls as boundary conditions rather than a general safety conclusion.

RF Safe presents itself as an RF exposure advocacy and education project promoting “RF exposure literacy,” safer-use habits, and updated safety frameworks beyond thermal-only assumptions. The post highlights RF Safe’s tools, including a SAR comparison database based on FCC SAR data, a public research viewer described as containing 4,000+ peer-reviewed studies, and its “TruthCase”/editorial standards. It argues that non-thermal biological interactions are reported in experimental literature and that compliance with current SAR limits does not necessarily reflect optimal real-world exposure outcomes.

RF Safe publishes a rebuttal to Media Bias Fact Check’s (MBFC) decision to rate the site “Medium Credibility,” addressing MBFC’s concerns about selective citation, one-sided interpretation, alarmist framing, and potential conflicts of interest tied to selling RF-safety products. The post argues RF Safe includes null/negative findings, avoids claiming RF “causes” specific diseases, and maintains editorial/transparency policies meant to separate evidence types and disclose commercial relationships. It also contends MBFC’s critique is partly a dispute over tone and wording (e.g., “primarily” funded by product sales) rather than demonstrated sourcing errors.

RF Safe argues that “no effect” (null) findings in RF research should be treated as informative constraints rather than dismissed, within its S4–Mito–Spin mechanistic framework. The post claims biological and exposure heterogeneity can produce nonlinear, tissue- and signal-dependent outcomes, making null results an expected pattern under many study conditions. It references a WHO-commissioned systematic review on RF-EMF and oxidative stress biomarkers as concluding the evidence is of “very low certainty,” citing bias, heterogeneity, and exposure/measurement limitations.

This PubMed-listed paper argues that the U.S. regulatory framework for radiofrequency radiation (RFR) from wireless technologies is outdated, lacks adequate oversight and enforcement, and has not been meaningfully updated since 1996. It contends that FCC exposure limits focus on short-term, high-intensity effects and do not address long-term, low-intensity exposures, with insufficient safeguards for children, pregnancy, and other vulnerable groups. The authors also discuss alleged regulatory capture, gaps in monitoring and compliance, and propose reforms including independent research, updated safety limits, and stronger pre- and post-market surveillance.

RF Safe argues that celebrity-branded mobile services (citing reported plans for “Beast Mobile” and the announced “Trump Mobile”) could normalize near-body, all-day phone use—especially among children—and therefore carry ethical responsibility for scaled RF exposure. The piece cites legal and scientific developments (including the 2021 Environmental Health Trust v. FCC decision, the U.S. NTP animal studies, and a WHO-commissioned systematic review) to claim the evidence base has “moved decisively” toward concern about long-term RF-EMF effects. It also promotes a proposed mechanistic framework ("S4–Mito–Spin") and suggests shifting indoor connectivity toward Li‑Fi (IEEE 802.11bb) as a harm-reduction approach.

RF Safe argues that recent WHO-linked evidence reviews have moved beyond a “thermal-only” safety narrative and that policy should respond with stronger protections. The post cites a 2025 WHO-commissioned systematic review in Environment International as concluding with “high certainty” that RF-EMF increases malignant heart schwannomas and brain gliomas in male rats, and references a 2025 corrigendum upgrading certainty for reduced pregnancy rates after male RF exposure in animal experiments. It also points to U.S. FCC rules being rooted in 1996-era assumptions and references a U.S. appellate court remand requiring the FCC to better address non-cancer harms and impacts on children and long-term exposure. The article advocates for the “Clean Ether Act” and promotes RF Safe’s proposed “S4–Mito–Spin” mechanism framework as a non-thermal explanatory model.

RF Safe promotes an “Act Now” hub intended to convert EMF safety concerns into policy and regulatory actions, emphasizing accountability and exposure reduction, especially for children. The page outlines five advocacy “levers,” including changing Section 704 of the Telecommunications Act, pressing the FCC to complete actions following a court remand, and restarting a federal electronic-product radiation program. It frames current RF oversight as outdated and insufficient for modern exposure patterns, and provides scripts to help supporters submit comments and demands into official records.

This animal study tested whether short-term 28 GHz (5G-band) millimeter-wave exposure modifies doxorubicin-induced cardiotoxicity in male rats and whether vitamin C mitigates effects. Co-exposure to 28 GHz EMR was reported to worsen several indices of DOX-related cardiac injury (including CAT reduction, increased BAX expression, and QT prolongation), while vitamin C provided partial attenuation. The authors emphasize that results are limited to a short-duration preclinical model and that human relevance remains preliminary.

This in vitro study examined strictly non-thermal, GSM-like 3.5 GHz RF-EMF exposure in cultured mouse dorsal root ganglion neurons for 1–24 hours. The authors report time-dependent reductions in cell viability alongside increased ROS and changes consistent with mitochondria-mediated apoptosis (e.g., Bax/caspase-3 up, cytochrome c release, Bcl-2 down) and increased p75NTR. They conclude these findings provide mechanistic evidence of peripheral neuronal vulnerability to mid-band RF exposure and call for further in vivo research.

This RF Safe position piece argues that long-term exposure to “non-native,” low-fidelity electromagnetic environments (including man-made RF) can degrade biological timing and coherence, contributing to downstream issues such as immune dysregulation and oxidative stress. It frames this as a systems-level claim rather than asserting RF “causes” specific diseases, and it cites proposed biophysical mechanisms (e.g., coupling into dense tissues, membrane voltage-sensing domains, mitochondrial/redox pathways). The article also references Heinrich Hertz’s historical exposure to early radio experiments and a retrospective medical analysis of his illness, while stating it is not claiming RF caused his condition.

RF Safe argues that common marketing claims for anti-radiation phone cases (e.g., “99% shielding”) are misleading because they often rely on controlled lab fabric tests that do not reflect real-world phone use. The post claims factors like shield orientation, phone transmit-power increases under obstruction, frequency differences (including 5G bands), and user/body interactions can reduce or even reverse purported exposure reductions. It also criticizes current regulatory testing frameworks for not requiring phones to be tested with cases and promotes RF Safe’s own “TruthCase/QuantaCase” approach as a more honest alternative.