This narrative review discusses how environmental factors may affect male reproductive health through epigenetic mechanisms, including DNA methylation, histone modifications, chromatin remodeling, and non-coding RNA regulation. It reports that electromagnetic radiation, particularly from mobile phones and wireless devices, is linked in the reviewed literature to reduced sperm count and motility, increased oxidative stress, and chromatin damage. The authors conclude there is a substantive connection between EMF exposure and adverse male reproductive outcomes and suggest practical risk-reduction guidance.

This review discusses potential impacts of radiofrequency electromagnetic fields from technologies such as Wi‑Fi and mobile phones on the central nervous system, neurotransmitter dynamics, and reproductive health. It describes proposed mechanisms including oxidative stress, thermal effects, altered neurotransmitter activity, ion channel changes, and neuronal apoptosis, while acknowledging conflicting evidence. The authors note that Wi‑Fi RF exposure has not been confirmed to exceed safety guidelines but argue that updated standards and long-term studies are needed, particularly for children/adolescents and in the context of expanding technologies such as 5G.

This biophysics paper presents a nonequilibrium (active matter) statistical mechanics model for electromechanical biological membranes. It argues that energy-driven activity in membranes could enable detection of electric fields far below equilibrium thermal-noise limits, and reports that the model can reproduce experimental observations by tuning activity. The abstract frames this as a potential mechanistic link between weak electromagnetic fields and biological responses, while also noting future modeling directions and possible implications for exposure safety discussions.

In a rat experiment (n=59), a single 40-minute whole-body 28 GHz exposure at near-threshold WBA-SAR levels was evaluated under normal and heat conditions with restraint. After accounting for sham-related restraint stress, exposure was associated with increased serum-free corticosterone 1–3 days later, especially when rectal temperature rose by >1°C. Urinary catecholamines suggested an immediate inhibitory effect on stress response (notably noradrenaline), with heat amplifying effects and linking noradrenaline to tail surface temperature.

This animal study exposed Sprague-Dawley rats (including pregnant females and offspring) to 2.45 GHz Wi-Fi or mobile jammer radiation for 2 hours daily over two weeks and assessed thyroid hormones and thyroid histology. The abstract reports significant changes in T4 in exposed adult males and significant differences in T3 among male offspring exposed to jammer radiation. Histopathology reportedly showed disrupted thyroid follicular structure in exposed rats. The authors conclude these findings support a potential link between non-ionizing radiation exposure and altered thyroid endocrine and histological parameters.

This animal study exposed rats to a 1800 MHz electromagnetic field for 12 weeks and assessed hormones, sperm quality, and behavior. The abstract reports increased corticosterone, decreased thyroid-stimulating hormone, reduced sperm motility/viability, and increased anxiety-like behavior in exposed rats. Some hormonal changes reportedly persisted for at least 2 weeks after exposure ended, and the authors frame the results as indicating adverse endocrine, reproductive, and behavioral effects.

This animal study examined maternal mobile phone exposure during different gestational windows in Wistar rats and assessed ovarian development and hormones in neonatal offspring. Compared with sham (phone off), exposed groups (standby and conversation/standby) were reported to have lower neonatal estrogen and progesterone and reduced primordial follicle/primary oocyte measures, with stronger effects after longer exposure. The study also reports increased primordial follicle apoptosis, particularly in the conversation/standby condition, and notes effects even with first-week gestational exposure.

This occupational epidemiology study used machine learning to evaluate whether workplace exposures (including magnetic and electric fields, vibration, noise, and heat stress) predict male reproductive indicators in 80 workers. The models and explainable AI outputs highlighted magnetic and electric field exposures and age as key predictors linked to lower free testosterone. The authors also report a 10-year forecast identifying electric field exposure as the most important long-term risk factor.

This mini-review discusses indoor radiation sources that may affect companion animals, including radon, radionuclides in feed, radiofrequency sources (phones, Wi-Fi, pet tracking devices), solar radiation, and extremely low frequency radiation. It reports that indoor radiation may negatively impact companion animal health and well-being. The authors conclude that preventive and precautionary measures are necessary to protect companion animals from indoor radiation exposure.

This animal study reports that prolonged RF-EMR exposure (2.45 GHz for 8 weeks) increased oxidative stress and ferroptosis in mouse testicular tissue and was associated with reduced sperm quality. Melatonin administration reportedly mitigated oxidative injury and inhibited ferroptosis. The abstract attributes the protective effect to Nrf2 pathway activation via MT1/MT2 receptors.

This study reports that terahertz radiation decreased a neuronal membrane area ratio (cytosol relative to protruding membrane area) beginning on the first day of exposure and persisting during the exposure period. It further reports altered neuronal firing/discharge patterns and increased peak postsynaptic currents associated with the morphology change, supported by a kinetic model. The authors frame the findings as indicating significant effects of terahertz-frequency EMF on neural health and function and suggest potential neuromodulation applications.