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The Evidence Is Now Decisive: Man Made Radiofrequency Fields Can Cause Cancer and Other Serious Biological Harm – And We Finally Know Exactly How

Independent Voices RF Safe Nov 26, 2025

An RF Safe article argues that, as of 2025, evidence is “decisive” that man-made radiofrequency (RF) fields can cause cancer and other biological harm, and that non-thermal mechanisms are now established. It cites animal studies (including NTP and Ramazzini), a 2025 WHO-commissioned systematic review (as described by the author), and proposed mechanisms involving voltage-gated ion channels, oxidative stress, and radical-pair/spin chemistry. The piece calls for updated safety standards that consider modulation and tissue vulnerability, while stating it is “not a call for panic.”

Model Variability in Assessment of Human Exposure to Radiofrequency Fields

Research RF Safe Research Library Jan 1, 2025

This review examines how variability in computational dosimetry models affects assessment of human RF exposure from MHz to terahertz frequencies, focusing on SAR, absorbed power density, and temperature rise. It reports that anatomical scaling and model choices can drive meaningful differences in predicted SAR (including higher values in children/smaller models), while temperature-rise predictions are especially sensitive to thermophysiological parameters and vascular modeling. The authors indicate that computed variability remains within ICNIRP/IEEE safety margins but argue that uncertainties warrant ongoing research and refinement as new technologies (e.g., 6G) emerge.

5G Radiofrequency Exposure Reduces PRDM16 and C/EBP � mRNA Expression, Two Key Biomarkers for Brown Adipogenesis

Research RF Safe Research Library Jan 1, 2025

This animal study exposed juvenile and young adult Wistar rats to 5G (3.5 GHz) or 2G (900 MHz) radiofrequency fields (1.5 V/m) for 1–2 weeks and measured brown adipose tissue-related gene expression by RT-qPCR. The abstract reports significant downregulation of PRDM16 and C/EBP mRNA after 5G exposure, while UCP1-dependent thermogenesis markers were not significantly changed at the transcriptional level. The authors interpret these findings as a potential partial disruption of brown adipocyte differentiation and raise EMF safety concerns, while calling for further confirmatory research.

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