This RF Safe article argues that pulsed, low-frequency-modulated wireless radiofrequency exposures could disrupt voltage-gated ion channel timing (via the S4 voltage sensor), leading to altered immune-cell signaling, mitochondrial oxidative stress, and downstream innate immune activation and inflammation. It presents a mechanistic narrative linking small membrane-potential shifts to changes in calcium and proton channel behavior, then to mitochondrial reactive oxygen species and inflammatory pathways (e.g., cGAS–STING, TLR9, NLRP3). The post cites animal findings and a described 2025 mouse gene-expression study as supportive, but the piece itself is not a peer-reviewed study and some claims are presented as deterministic without providing full methodological details in the excerpt.