This RF Safe commentary argues that wireless communications “took a wrong turn” by prioritizing radiofrequency/microwave transmission over light-based approaches, citing Alexander Graham Bell’s 1880 photophone as an alternative model. It suggests that widespread, continuous RF exposure in modern environments is undesirable and proposes light-based, room-scale wireless as more biologically compatible. The piece also speculates about a historical association between Heinrich Hertz’s close-range RF experiments and his later fatal illness, while acknowledging there is no controlled evidence proving causation.

An RF Safe commentary argues that the U.S. Department of Health and Human Services (HHS) is violating Public Law 90-602 by failing to continuously update radiation-safety standards, asserting that no formal revisions have occurred since the mid-1980s. The post links this alleged inaction to continued public exposure from wireless technologies and calls for renewed long-term research and stricter exposure limits. It also claims the National Toxicology Program (NTP) was shut down in 2024 and references a 2021 court decision criticizing FCC RF rules, urging congressional action and new legislation.

This RF Safe commentary proposes a speculative “bioelectric hypothesis” for the Fermi paradox: that widespread, continuous use of man-made radiofrequency/microwave emissions (“hertzification”) could act as a slow “Great Filter” that causes technological civilizations to decline or go silent. The author argues that modern RF environments create an unprecedented, omnipresent exposure for organisms and suggests potential biological vulnerability via voltage-gated ion channels. The piece is framed as an exploration rather than a reported study and does not present new empirical data in the provided excerpt.

This animal study used Drosophila knockout lines to examine whether visual (Rh1) versus non-visual (Rh7) opsins contribute to blue-light-associated neural damage. Flies were continuously exposed to 488 nm blue light from egg deposition to 20 days, and brain DNA damage and vacuolisation were assessed. The study reports greater DNA damage and neurodegeneration markers in Rh1 knockout flies than in wild-type or Rh7 knockout flies, and concludes Rh1 is a predominant mediator of blue-light-induced neurotoxicity in the fly CNS.

This animal experimental study exposed chicken embryos (CAM) continuously to 2.4 GHz Wi-Fi at an average power density of 300 μW/m2 for 9 or 14 embryonic days. H&E staining reportedly showed no significant structural differences in large vessel walls versus controls. However, special staining reported decreased optical density of elastic fibers at both time points and changes in collagen fiber optical density (increase at day 9, decrease at day 14). The authors conclude Wi-Fi exposure can alter fibrous vessel wall components and suggest potential relevance to cardiovascular disorders.

This in vitro study used LC-MS metabolomics to assess how continuous versus intermittent RF-EMF irradiation affects mouse Leydig (TM3) and spermatogonia (GC-1) cells. The authors report stronger metabolic disturbances in TM3 cells under continuous exposure, including changes in amino acid and glutathione-related pathways, while intermittent exposure mainly affected fatty acyl and purine-related metabolism. GC-1 cells were reported to be less sensitive, and ADP changes were proposed as a potential metabolic signature. The authors interpret these metabolic disturbances as suggesting potential reproductive health risks.

This animal study examined whether continuous prenatal exposure to 2450 MHz radiofrequency radiation (simulating Wi‑Fi) affects thyroid tissue in rat offspring at 6 months. The exposed group showed significantly increased mononuclear cell infiltration and vascular congestion in thyroid histology. TUNEL-positive cell percentage and H2A.X antibody levels did not differ significantly between groups.

This rat study tested whether acute exposure to 0.9 and 1.8 GHz continuous-wave radiofrequency radiation alters blood-brain barrier permeability. Using Evans-blue/albumin as a tracer, the authors report no BBB leakage in exposed female rats but a significant increase in albumin in exposed male rat brains versus sham. The authors interpret this as suggesting BBB/vascular permeability changes in males at SAR levels stated to be below international limits.