A multicenter case-control study in Iran reported that self-reported prolonged mobile phone use was associated with higher odds of confirmed and suspected breast cancer status. The authors emphasize that the findings do not imply causation and note limitations including self-reported exposure and potential residual confounding. They call for larger prospective studies with objective exposure assessment.

This RF Safe position piece argues that long-term exposure to “non-native,” low-fidelity electromagnetic environments (including man-made RF) can degrade biological timing and coherence, contributing to downstream issues such as immune dysregulation and oxidative stress. It frames this as a systems-level claim rather than asserting RF “causes” specific diseases, and it cites proposed biophysical mechanisms (e.g., coupling into dense tissues, membrane voltage-sensing domains, mitochondrial/redox pathways). The article also references Heinrich Hertz’s historical exposure to early radio experiments and a retrospective medical analysis of his illness, while stating it is not claiming RF caused his condition.

RF Safe presents “TruthCase™ · Clean Ether Action Hub” as a combined product-and-policy hub arguing that evidence from multiple RF health research lines supports harm occurring below current exposure limits. It promotes a proposed “S4–Mito–Spin / IFO‑VGIC” framework and a “density-gated” vulnerability map, and calls for policy actions such as changes to Section 704 and enforcement via FDA/FTC. The page frames regulatory “capture/inertia” as a key reason current limits persist, while positioning its view as a “respectable minority” in 2025.

RF Safe publishes a commentary describing a public feud between Dr. Jack Kruse and RF Safe founder John Coates over how to address health concerns attributed to non-native electromagnetic fields (nnEMFs), especially regarding children. The piece portrays Kruse as emphasizing personal “light/circadian” biohacks and Coates as pushing technology and policy changes such as LiFi adoption and repealing/altering telecom-related legal constraints. It includes numerous claims about EMF-related harms and references to research (e.g., NTP/Ramazzini, a Henry Lai meta-analysis) but presents them within an advocacy narrative rather than as a balanced review.

RF Safe presents the “S4-Mito-Spin” framework as a hypothesis aiming to unify proposed non-thermal biological effects reported in some EMF studies (e.g., oxidative stress, DNA damage, fertility effects, and tumors in animal models). The article describes a multi-mechanism model involving voltage-gated channel forced oscillation, mitochondrial/NOX amplification to reactive oxygen species bursts, and radical-pair/spin-state effects, with a novel “density-gated” concept to explain tissue-specific and inconsistent findings. It also suggests the framework could connect EMF hazards with therapeutic uses, citing FDA-approved RF devices such as TheraBionic as an example of RF modulation of biology.

This multicenter case-control study in Iran (n=226) examined associations between mobile phone use and breast cancer outcomes in women. Reporting more than 60 minutes/day of phone conversations was associated with higher odds of confirmed invasive breast cancer and of being classified as a suspected case versus <10 minutes/day. The authors emphasize that the results do not establish causation and may be influenced by self-reported exposure and residual confounding, warranting cautious interpretation.

This narrative review examines research on radiofrequency electromagnetic radiation (RF-EMR) from mobile towers and its potential association with genetic damage and genetic polymorphism in humans, with an emphasis on India. The abstract states that RF-EMR exposure may affect genetic material and suggests a link between EMR exposure and genetic damage, with possible implications for cancer risk and cell death. It also highlights that genetic polymorphisms may modify susceptibility and calls for further research to clarify health impacts.

This prospective cohort study followed 105 neonates/infants for one year and measured household RF-EMF using a selective radiation meter, categorizing exposure into tertiles. Higher household RF-EMF exposure was associated with lower ASQ-3 neurodevelopmental scores, particularly in motor and problem-solving domains, and higher odds of monitor/refer classifications for fine motor and problem-solving. The abstract notes these associations persisted after adjustment for low birth weight, though exposure was measured at a single time point and key confounders (e.g., prenatal phone use, parental interaction) were not assessed.

This retrospective observational study analyzed Murmuras app data from 1074 participants in 2022 to examine demographic differences in smartphone use and nocturnal smartphone inactivity duration (a proxy for sleep-related behavior). Nighttime smartphone use increased, especially for social media and entertainment, and usage patterns varied by gender, age, education, and employment status. Most demographic groups showed no significant correlation between usage duration and nocturnal inactivity, although some subgroups showed correlations in either direction. The authors frame excessive nighttime smartphone use as potentially adverse for sleep and link this behavioral exposure to electromagnetic fields with sleep health risks.

This exposure assessment measured RF power density across different floors of a high-rise university building using a spectrum analyser and log-periodic antenna. Power density decreased from the ground to the third floor but peaked on the fourth (top) floor. The 900 MHz band showed the highest reported power density (1.16E-03 W/m2), and the authors suggest higher-floor occupants may experience higher RF exposure from nearby communication towers.

This animal study reports that dual-frequency RF EMR exposure (0.8/2.65 GHz, 4 W/kg) induced anxiety-like behavior in mice. It also reports reduced CB1R and endocannabinoid levels in the mPFC and altered endocannabinoid system markers in the BLA. CB1R overexpression or knockdown in the mPFC reportedly decreased or increased anxiety-like behavior, respectively, suggesting a mechanistic link in this model.