This RF Safe article argues that biological effects from radiofrequency and pulsed electromagnetic fields can be interpreted through two complementary layers: Maxwell–Wagner interfacial polarization (as a direct electrodynamic mechanism at cell membranes) and an “S4–Mito-Spin” framework (as an upstream susceptibility model tied to voltage-sensor density, mitochondrial coupling, and antioxidant buffering). It suggests these mechanisms could converge on outcomes such as altered red-blood-cell stability, blood rheology, membrane deformation, and—at higher intensities—electroporation or hemolysis. The piece is presented as a mechanistic synthesis rather than reporting new experimental results, and it frames potential vulnerability to pulsed/non-native exposures as context-dependent.

This RF Safe article discusses rouleaux formation (reversible red blood cell stacking) and proposes a speculative mechanism by which weak magnetic fields might influence red blood cell surface charge (zeta potential) via spin chemistry in heme-related radical-pair processes. The piece frames the idea as a mechanistic “what if?” rather than a direct claim that everyday phone use causes blood clotting, and it leans on general concepts from hematology and radical-pair magnetosensitivity (e.g., cryptochrome in animals). No new experimental data are presented in the provided text; the argument is largely theoretical and interpretive.

In a rat experiment (n=59), a single 40-minute whole-body 28 GHz exposure at near-threshold WBA-SAR levels was evaluated under normal and heat conditions with restraint. After accounting for sham-related restraint stress, exposure was associated with increased serum-free corticosterone 1–3 days later, especially when rectal temperature rose by >1°C. Urinary catecholamines suggested an immediate inhibitory effect on stress response (notably noradrenaline), with heat amplifying effects and linking noradrenaline to tail surface temperature.