This animal study examined whether paternal exposure to 4.9 GHz (5G) radiofrequency radiation affects male offspring in C57BL/6 mice. It reports increased anxiety-like behavior and reduced sperm quality in adult F1 males from exposed fathers, alongside reported LRGUK hypermethylation and reduced LRGUK expression in testes. The abstract reports no significant effects on depression-like behavior, learning/memory, or fertility across F1–F2 generations.

This rat study assessed 30-day 2600 MHz EMF exposure effects on kidney tissue and DNA damage in blood lymphocytes, with an EMF+quercetin group included. Kidney histopathology and immunohistochemistry were reported as similar across groups, and oxidative stress markers did not significantly change. The EMF-only group showed significant DNA damage in lymphocytes by Comet assay.

This animal study exposed Aedes aegypti larvae to 5G-NR RF-EMF at 3.6 GHz for 5 days under two feeding regimes. The study reports delayed development at a lower exposure level mainly in nutritionally weakened larvae, and at a higher exposure level reports developmental changes and reduced adult size attributed to dielectric heating. Mortality and wing length asymmetry were reported as unchanged, and the authors note such high exposure levels are unlikely in natural aquatic settings.

This animal behavioral study observed 36 domestic dogs to assess whether magnetic fields from high-voltage power lines influence dogs' geomagnetic alignment behavior. Dogs showed bimodal alignment under control conditions and under north-south oriented power lines, but alignment became trimodal under east-west oriented lines with statistically significant differences versus control. The authors interpret these findings as indicating that power-line-related fields can alter orientation behavior and frame this as supporting concern about biological effects of EMF exposure.

This review discusses how increasing ambient nonionizing EMF (0–300 GHz), particularly RF from modern wireless technologies and satellites, may affect flora and fauna at ecosystem levels. It states that many nonhuman species rely on electro/magneto-reception and that even low-intensity EMF exposures are capable of disrupting critical biological functions and behaviors. The authors conclude that current exposure standards focus on human health and recommend policy reforms and mitigation measures to protect wildlife and ecosystems.

This two-sample Mendelian randomization study examined whether duration of mobile phone use is causally related to aneurysmal subarachnoid hemorrhage using large European GWAS datasets. The primary MR analysis reported that excessive duration of mobile phone use was associated with higher aSAH risk, and sensitivity analyses were described as supportive. The authors conclude the findings have potential clinical, public health, and policy implications.

This multicenter case-control study in Iran (n=226) examined associations between mobile phone use and breast cancer outcomes in women. Reporting more than 60 minutes/day of phone conversations was associated with higher odds of confirmed invasive breast cancer and of being classified as a suspected case versus <10 minutes/day. The authors emphasize that the results do not establish causation and may be influenced by self-reported exposure and residual confounding, warranting cautious interpretation.

No abstract was provided. From the title and supplied overview, this paper critiques the Mevissen et al. systematic review on RF-EMF exposure and cancer in animal studies, asserting that methodological and analytical flaws led to misjudgment of the evidence. The provided text frames the topic as requiring careful analysis to avoid underestimating potential health risks.

This magazine article reviews the Japan-Korea "NTP Lite" RF animal toxicity collaboration and its relationship to prior NTP (2018) and Ramazzini Institute reports of RF-associated tumors in male rats. It notes NTP Lite used a single whole-body SAR of 4 W/kg and completed a two-year exposure phase in 2022, but final reporting is delayed with histopathology and genotoxicity work ongoing. The author highlights protocol harmonization across labs while raising concerns about unexplained animal deaths and physiological differences in exposed groups, and frames the broader evidence as supportive of RF-related cancer risk in laboratory animals.

This article reviews the microwave auditory effect (perceived clicks/buzzing) that can occur when the head is exposed to pulsed microwave energy, such as from radar. It explores whether this phenomenon could plausibly explain reported “anomalous health incidents” (Havana syndrome), noting that experts and formal panels have suggested it as a possible explanation. The authors emphasize that potential links between pulsed microwave exposures, audible sensations, and other physiological impacts warrant careful consideration and further research.

This animal study exposed male Sprague Dawley rats to 2.45 GHz Wi-Fi for varying daily durations over eight weeks and assessed reproductive hormones and LHCGR expression. Serum LH and testosterone did not differ significantly from controls, but LHCGR mRNA increased with longer exposure and LHCGR protein showed decreases with shorter exposures with partial improvement at 24 hours/day. The findings suggest molecular alterations in testicular tissue despite stable systemic hormone levels.

This animal study examined Wi-Fi RF-EMR exposure in adult zebrafish (4 hours/day for 30 days) and assessed reproductive tissues and offspring outcomes. The abstract reports testicular and ovarian histopathological abnormalities in exposed adults. Offspring from exposed parents, maintained under EMF-free conditions, reportedly showed increased mortality, morphological abnormalities, and anxiety-like behavior, with malformations increasing with longer parental exposure.

This animal study used Drosophila knockout lines to examine whether visual (Rh1) versus non-visual (Rh7) opsins contribute to blue-light-associated neural damage. Flies were continuously exposed to 488 nm blue light from egg deposition to 20 days, and brain DNA damage and vacuolisation were assessed. The study reports greater DNA damage and neurodegeneration markers in Rh1 knockout flies than in wild-type or Rh7 knockout flies, and concludes Rh1 is a predominant mediator of blue-light-induced neurotoxicity in the fly CNS.